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AAC Accepts, published online ahead of print on 24 November 2008
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Antimicrob. Agents Chemother. doi:10.1128/AAC.00132-08
Copyright (c) 2008, American Society for Microbiology and/or the Listed Authors/Institutions. All Rights Reserved.

SOS Regulation of qnrB Expression

Minghua Wang, George A. Jacoby*, Debra M. Mills, and David C. Hooper

Massachusetts General Hospital, Boston, Massachusetts; Institute of Antibiotics, Huashan Hospital, Fudan University, Shanghai, China; and Lahey Clinic, Burlington, Massachusetts

* To whom correspondence should be addressed. Email: george.a.jacoby{at}lahey.org.


  Abstract

In the sequence upstream from qnrB (but not qnrA or qnrS) is a LexA binding site. qnrB was shown to be under SOS control by demonstrating that quinolone susceptibility decreased with increasing temperature in a strain with a recA441 (ts) allele whereas qnrB expression increased in response to ciprofloxacin or mitomycin C in strains with an intact LexA gene.






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